| 1) C than CWF (77·5% versus 59·8% migrated cells). |
| 2) closely respected the microenvironment of cells. |
| 3) air mechanisms carried out by endothelial cells. |
| 4) ocytes that can potentially kill melanoma cells. |
| 5) terferes with the differentiation of K562 cells. |
| 6) sion and interactions of neural and other cells. |
| 7) onally, apoptosis occurred in PDT-treated cells. |
| 8) s, and focal infiltration of inflammatory cells. |
| 9) P < 0.05) of cell growth rate in MCF-7 cells. |
| 10) fferentiation potential than FCS-expanded cells. |
| 11) e the primary effect of NaVO(3) on CHO-K1 cells. |
| 12) to inhibit proliferation of human cancer cells. |
| 13) p-PERK, MMP-2, MMP-9, and VEGF in U-2 OS cells. |
| 14) and invasion of U-2 OS human osteosarcoma cells. |
| 15) portant biomarkers to assess apoptosis in cells. |
| 16) growth, differentiation and apoptosis of cells. |
| 17) apamycin was observed in human and canine cells. |
| 18) an in human leukaemia and canine lymphoma cells. |
| 19) ose and time-dependent signicants in A549 cells. |
| 20) psulomas with the resident renal capsular cells. |
| 21) nt way for large-scale expansion of these cells. |
| 22) interactions between adjacent epithelial cells. |
| 23) s due to the prolonged engraftment of UCB cells. |
| 24) within the mucosal epithelial basal layer cells. |
| 25) cin within the retinal pigment epithelial cells. |
| 26) ed by TRPV1 activation in MCF-7 and CF.41 cells. |
| 27) L-6) were significantly higher in treated cells. |
| 28) expression and protein levels in SK-Hep1 cells. |
| 29) e complex had an apoptotic effect on HeLa cells. |
| 30) n (PI3K/AKT) in human and canine melanoma cells. |
| 31) dium constituents, well plate plastic and cells. |
| 32) revealed RANKL expression by CD44+ cancer cells. |
| 33) ages, astrocytes and vascular endothelial cells. |
| |